716 research outputs found
Diabetic mouse angiopathy is linked to progressive sympathetic receptor deletion coupled to an enhanced caveolin-1 expression.
- Author
- Publication venue
- 'Ovid Technologies (Wolters Kluwer Health)'
- Publication date
- 01/01/2004
- Field of study
OBJECTIVE:
Clinical studies have demonstrated that hyperglycaemia represents a major risk factor in the development of the endothelial impairment in diabetes, which is the first step in vascular dysfunction. Using non-obese diabetic mice, we have evaluated the role of the adrenergic system and eNOS on progression of the disease
METHODS AND RESULTS:
When glycosuria is high (20 to 500 mg/dL), there is a selective reduction in the response to alpha1 and beta2 agonists but not to dopamine or serotonin. When glycosuria is severe (500 to 1000 mg/dL), there is a complete ablation of the contracture response to the alpha1 receptor agonist stimulation and a marked reduced response to beta2 agonist stimulation. This effect is coupled with a reduced expression of alpha1 and beta2 receptors, which is caused by an inhibition at transcriptional level as demonstrated by RT-PCR. In the severe glycosuria (500 to 1000 mg/dL), although eNOS expression is unchanged, caveolin-1 expression is significantly enhanced, indicating that high glucose plasma levels cause an upregulation of the eNOS endogenous inhibitory tone. These latter results correlate with functional data showing that in severe glycosuria, there is a significant reduction in acetylcholine-induced vasodilatation.
CONCLUSIONS:
Our results show that in diabetes development, there is a progressive selective downregulation of the alpha1 and beta2 receptors. At the same time, there is an increased expression of caveolin-1, the endogenous eNOS inhibitory protein. Thus, caveolin-1 could represent a new possible therapeutic target in vascular impairment associated with diabetes
Search for direct stau production in events with two hadronic tau-leptons in root s=13 TeV pp collisions with the ATLAS detector
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- Q. Li
- Q. Ouyang
- Q. Wang
- Q. 
- R. Araujo Pereira
- R. Astalos
- R. Avramidou
- R. Barnea
- R. Bartoldus
- R. Bi
- R. Bielski
- R. Brenner
- R. Brock
- R. Camacho Toro
- R. Caminal Armadans
- R. Cardarelli
- R. Castelijn
- R. Cherkaoui El Moursli
- R. De Asmundis
- R. Di Nardo
- R. Di Sipio
- R. El Kosseifi
- R. Ferrari
- R. Gamboa Goni
- R. Gonç
- R. Goncalves Gama
- R. Gugel
- R. Gupta
- R. Hankache
- R. Hauser
- R. Hertenberger
- R. Hyneman
- R. Ignazzi
- R. Iguchi
- R. Iuppa
- R. Jansky
- R. Keeler
- R. Kehoe
- R. Konoplich
- R. Kopeliansky
- R. Kowalewski
- R. Kukla
- R. Lafaye
- R. Leitner
- R. Leone
- R. Les
- R. Lysak
- R. Madar
- R. Mashinistov
- R. Mazini
- R. Moles-Valls
- R. Narayan
- R. Nicolaidou
- R. Nisius
- R. Novotny
- R. Ospanov
- R. Pedro
- R. Pezoa
- R. Piegaia
- R. Poettgen
- R. Poggi
- R. Polifka
- R. Rö
- R. Richter
- R. Rosten
- R. Sadykov
- R. Sawada
- R. Schwienhorst
- R. Shang
- R. Simoniello
- R. Slovak
- R. Sobie
- R. Soualah
- R. Spighi
- R. Stamen
- R. Staszewski
- R. Strö
- R. Stroynowski
- R. Tafirout
- R. Takashima
- R. Tanaka
- R. Teixeira De Lima
- R. Vari
- R. Vuillermet
- R. Walker
- R. Wang
- R. Wang
- R. Wolff
- R. Yuan
- R. Zhang
- R. Zou
- R. 
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- R.-. Wang
- S. Adachi
- S. Adorni
- S. Akatsuka
- S. Alderweireldt
- S. Amoroso
- S. Angelidakis
- S. Argyropoulos
- S. Artz
- S. Asai
- S. Balaji
- S. Banerjee
- S. Barsov
- S. Batlamous
- S. Bentvelsen
- S. Berlendis
- S. Bethke
- S. Biondi
- S. Bressler
- S. Bruno
- S. Burdin
- S. Cabrera Urbá
- S. Calvente Lopez
- S. Calvet
- S. Camarda
- S. Campana
- S. Caron
- S. Carrá
- S. Che
- S. Chekanov
- S. Chen
- S. Chouridou
- S. Constantinescu
- S. Cré
- S. Czekierda
- S. Dí
- S. D’
- S. Dahbi
- S. De Castro
- S. De Cecco
- S. Demers
- S. Dysch
- S. Falciano
- S. Falke
- S. Farrell
- S. Francescato
- S. Franchino
- S. Gadatsch
- S. Ganguly
- S. Gargiulo
- S. Gentile
- S. George
- S. Ghasemi
- S. Giagu
- S. Gkaitatzis
- S. Goldfarb
- S. Gonzá
- S. Gonzalez-Sevilla
- S. Grancagnolo
- S. Grinstein
- S. Groh
- S. Guindon
- S. Gurbuz
- S. Hagebö
- S. Han
- S. Harkusha
- S. Hassani
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- S. Heer
- S. Heim
- S. Hellesund
- S. Hellman
- S. Henkelmann
- S. Higashino
- S. Hirose
- S. Honda
- S. Hou
- S. Hu
- S. Hyrych
- S. Istin
- S. Jé
- S. Jakobsen
- S. Jiggins
- S. Jin
- S. Jones
- S. Kersten
- S. Ketabchi Haghighat
- S. Kido
- S. Kluth
- S. Koperny
- S. Kortner
- S. Kuday
- S. Kuehn
- S. Kuleshov
- S. Lai
- S. Lammers
- S. Laplace
- S. Leone
- S. Li
- S. Loffredo
- S. Maeland
- S. Majewski
- S. Maltezos
- S. Malyukov
- S. Manzoni
- S. Marti-Garcia
- S. Martin-Haugh
- S. Meehan
- S. Mehlhase
- S. Menke
- S. Mergelmeyer
- S. Mohapatra
- S. Monzani
- S. Morgenstern
- S. Muanza
- S. Nemecek
- S. Oda
- S. Odaka
- S. Oerdek
- S. Pagan Griso
- S. Palazzo
- S. Palestini
- S. Parajuli
- S. Passaggio
- S. Pataraia
- S. Perrella
- S. Popa
- S. Pospisil
- S. Prell
- S. Prince
- S. Protopopescu
- S. Rajagopalan
- S. Raspopov
- S. Rave
- S. Resconi
- S. Rettie
- S. Richter
- S. Rodriguez Bosca
- S. Roe
- S. Rosati
- S. Sacerdoti
- S. Saha
- S. Schaepe
- S. Schier
- S. Schlenker
- S. Schmitt
- S. Schmitz
- S. Schramm
- S. Sen
- S. Senkin
- S. Shimizu
- S. Shirabe
- S. Shrestha
- S. Simion
- S. Simsek
- S. Snyder
- S. Sottocornola
- S. Spagnolo
- S. Stä
- S. Stonjek
- S. Strandberg
- S. Suchek
- S. Sultansoy
- S. Sun
- S. Suzuki
- S. Tapia Araya
- S. Tapprogge
- S. Tarem
- S. Terada
- S. Terzo
- S. Timoshenko
- S. Tisserant
- S. Todorova-Nova
- S. Todt
- S. Toká
- S. Trincaz-Duvoid
- S. Tsuno
- S. Turchikhin
- S. Tzamarias
- S. Valentinetti
- S. Veneziano
- S. Wahdan
- S. Webb
- S. Wenig
- S. Williams
- S. Willocq
- S. Xella
- S. Yacoob
- S. Yang
- S. Ye
- S. Zambito
- S. Zimmermann
- S. 
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- S.-. Hsu
- T. Š
- T. Ž
- T. Adye
- T. Alexopoulos
- T. Andeen
- T. Barillari
- T. Barklow
- T. Beau
- T. Berry
- T. Bisanz
- T. Blazek
- T. Bold
- T. Buanes
- T. Cao
- T. Carli
- T. Cuhadar Donszelmann
- T. Dado
- T. Dai
- T. Daubney
- T. Davidek
- T. Dias Do Vale
- T. Djobava
- T. Dreyer
- T. Eifert
- T. Ekelof
- T. Farooque
- T. Flick
- T. Fusayasu
- T. Geralis
- T. Golling
- T. Guillemin
- T. Harenberg
- T. Heim
- T. Herrmann
- T. Hryn’
- T. Iizawa
- T. Jakoubek
- T. Javů
- T. Kaji
- T. Kawaguchi
- T. Kawamoto
- T. Kharlamova
- T. Kishimoto
- T. Klapdor-Kleingrothaus
- T. Klingl
- T. Klioutchnikova
- T. Kobayashi
- T. Koffas
- T. Koi
- T. Komarek
- T. Kondo
- T. Kono
- T. Kosek
- T. Kubota
- T. Kuhl
- T. Kunigo
- T. Kupfer
- T. Kwan
- T. Lagouri
- T. Lari
- T. LeCompte
- T. Lenz
- T. Lohse
- T. Lyubushkina
- T. Maeno
- T. Maier
- T. Mashimo
- T. Masubuchi
- T. Megy
- T. Meideck
- T. Mitani
- T. Mkrtchyan
- T. Moa
- T. Moskalets
- T. Nakamura
- T. Naumann
- T. Nguyen Manh
- T. Nitta
- T. Nobe
- T. Novak
- T. Okuyama
- T. Pauly
- T. Peiffer
- T. Pham
- T. Poulsen
- T. Rashid
- T. Saito
- T. Scanlon
- T. Seiss
- T. Sumida
- T. Swirski
- T. Sykora
- T. Takeshita
- T. Tashiro
- T. Theveneaux-Pelzer
- T. Trefzger
- T. Varol
- T. Vazquez Schroeder
- T. Vickey
- T. Wenaus
- T. Wengler
- T. Xu
- T. Yamazaki
- T. Zakareishvili
- T. 
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- T. 
- U. Blumenschein
- U. De Sanctis
- U. Gul
- U. Klein
- U. Kruchonak
- U. Landgraf
- U. Mallik
- U. Parzefall
- U. Schä
- U. Schnoor
- U. Soldevila
- V. Andrei
- V. Araujo Ferraz
- V. Bü
- V. Boisvert
- V. Canale
- V. Castillo Gimenez
- V. Cavaliere
- V. Cavasinni
- V. Chiarella
- V. Cindro
- V. Croft
- V. D’
- V. Dao
- V. Ellajosyula
- V. Fabiani
- V. Garonne
- V. Gratchev
- V. Hedberg
- V. Herget
- V. Ippolito
- V. Izzo
- V. Jain
- V. Kartvelishvili
- V. Kitali
- V. Konstantinides
- V. Kouskoura
- V. Kukhtin
- V. Lyubushkin
- V. Magerl
- V. Morisbak
- V. Nikolaenko
- V. O’
- V. Pleskot
- V. Polychronakos
- V. Sinetckii
- V. Smakhtin
- V. Solovyev
- V. Tsiskaridze
- V. Tsulaia
- V. Tudorache
- V. Vacek
- V. Vecchio
- V. Vercesi
- V. Vorobel
- V. Vrba
- V. Wallangen
- V. Zhulanov
- V. 
- V. 
- V. 
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- V. 
- V. 
- W. Buttinger
- W. Dabrowski
- W. Davey
- W. Ding
- W. Fedorko
- W. Guan
- W. Guo
- W. Islam
- W. Kozanecki
- W. Lampl
- W. Song
- W. Taylor
- W. Trischuk
- W. Vandelli
- W. Verkerke
- W. Wagner
- W. Walkowiak
- W. Wang
- W. Wiedenmann
- W. Xu
- W. Yao
- W. 
- W. 
- W. 
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- X. Ai
- X. Chen
- X. Chu
- X. Hoad
- X. Ju
- X. Li
- X. Lou
- X. Lou
- X. Ruan
- X. Sun
- X. Wu
- X. Xiao
- X. Yang
- X. Yue
- X. Zhang
- X. Zhuang
- X. 
- Y. Abulaiti
- Y. Afik
- Y. Amaral Coutinho
- Y. Benhammou
- Y. Cao
- Y. Chen
- Y. Chen
- Y. Coadou
- Y. Delabat Diaz
- Y. Duan
- Y. Enari
- Y. Fang
- Y. Fang
- Y. Gao
- Y. Guo
- Y. Hasegawa
- Y. Heng
- Y. Herná
- Y. Horii
- Y. Huang
- Y. Huang
- Y. Ikegami
- Y. Jiang
- Y. Kano
- Y. Kulchitsky
- Y. Li
- Y. Liu
- Y. Ma
- Y. Makida
- Y. Minegishi
- Y. Nagasaka
- Y. Nakahama
- Y. Ninomiya
- Y. Noguchi
- Y. Okazaki
- Y. Okumura
- Y. Qin
- Y. Rozen
- Y. Sano
- Y. Shen
- Y. Shimogama
- Y. Smirnov
- Y. Takubo
- Y. Tayalati
- Y. Tian
- Y. Tu
- Y. Unno
- Y. Wang
- Y. Wu
- Y. Yamaguchi
- Y. Yamazaki
- Y. Yang
- Y. Yasu
- Y. Zhang
- Y. Zhao
- Y. Zhou
- Y. Zhu
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Y. 
- Z. Barnovska-Blenessy
- Z. Chadi
- Z. Dolezal
- Z. Guo
- Z. Hubacek
- Z. Idrissi
- Z. Jiang
- Z. Li
- Z. Liang
- Z. Marshall
- Z. Rurikova
- Z. Uysal
- Z. Wang
- Z. Xi
- Z. Xu
- Z. Xu
- Z. Yan
- Z. Zhang
- Z. Zhang
- Z. Zhao
- Z. Zheng
- Z. Zinonos
- Z. 
- Z. 
- Z. 
- Z. 
- Z. 
- Z. 
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2020
- Field of study
A search for the direct production of the supersymmetric partners ofτ-leptons (staus) in final stateswith two hadronically decayingτ-leptons is presented. The analysis uses a dataset of pp collisions corresponding to an integrated luminosity of139fb−1, recorded with the ATLAS detector at the LargeHadron Collider at a center-of-mass energy of 13 TeV. No significant deviation from the expected StandardModel background is observed. Limits are derived in scenarios of direct production of stau pairs with eachstau decaying into the stable lightest neutralino and oneτ-lepton in simplified models where the two staumass eigenstates are degenerate. Stau masses from 120 GeV to 390 GeV are excluded at 95% confidencelevel for a massless lightest neutralino
Mitochondrial Superoxide Contributes to Blood Flow and Axonal Transport Deficits in the Tg2576 Mouse Model of Alzheimer's Disease
- Author
- B Halliwell
- B Jeynes
- BC Kone
- C Behl
- C Behl
- C Iadecola
- C Muscoli
- CA Massaad
- CD Smith
- CX Gong
- Cynthia A. Massaad
- D Fulton
- D Harman
- D Hu
- DA Butterfield
- DA Butterfield
- ED Roberson
- Eric Klann
- F Li
- FG De Felice
- G Amadoro
- G Lindwall
- GB Stokin
- HK Shin
- I Casserly
- I Grundke-Iqbal
- J Hardy
- J Vasquez-Vivar
- JC de la Torre
- K Hsiao
- KD Smith
- L Esposito
- L Park
- Lingyun Hu
- LM Ittner
- M Anantharaman
- M Dumont
- M Guglielmotto
- M Higuchi
- M Manczak
- MA Marletta
- MD Weingarten
- ME Calhoun
- MI Lin
- O Hansson
- OM Tajes
- P Wenzel
- R Christie
- RN Kalaria
- Robia G. Pautler
- S Dimmeler
- S Melov
- S Moncada
- Samir K. Amin
- SB Shah
- SP Gabbita
- Sérgio Teixeira Ferreira
- T Kawarabayashi
- TJ McCabe
- WC Sessa
- Y Xia
- YS Ho
- Yuan Mei
- Publication venue
- Public Library of Science
- Publication date
- 01/05/2010
- Field of study
Alzheimer's disease (AD) is a neurodegenerative disease characterized by the progressive decline in cognitive functions and the deposition of aggregated amyloid beta (Abeta) into senile plaques and the protein tau into tangles. In addition, a general state of oxidation has long been known to be a major hallmark of the disease. What is not known however, are the mechanisms by which oxidative stress contributes to the pathology of AD.In the current study, we used a mouse model of AD and genetically boosted its ability to quench free radicals of specific mitochondrial origin. We found that such manipulation conferred to the AD mice protection against vascular as well as neuronal deficits that typically affect them. We also found that the vascular deficits are improved via antioxidant modulation of the endothelial nitric oxide synthase, an enzyme primarily responsible for the production of nitric oxide, while neuronal deficits are improved via modulation of the phosphorylation status of the protein tau, which is a neuronal cytoskeletal stabilizer.These findings directly link free radicals of specific mitochondrial origin to AD-associated vascular and neuronal pathology
The Brain's Router: A Cortical Network Model of Serial Processing in the Primate Brain
- Author
- A Allport
- A Allport
- A Bollimunta
- A Burkhalter
- A Del Cul
- A Newell
- A Osman
- A Pouget
- A Stemme
- A Treisman
- A Treisman
- A Zylberberg
- AD Baddeley
- Ariel Zylberberg
- B Brisson
- B Wyble
- B Wyble
- BA Olshausen
- BA Olshausen
- BJ Baars
- C Kranczioch
- C Kranczioch
- C Sergent
- CC Lo
- CC Lo
- CE Jahr
- CE Schroeder
- CE Schroeder
- CJ Bruce
- CN Olivers
- CN Olivers
- CW Telford
- D Heinke
- D Heinke
- D Heinke
- D Jilk
- D Zipser
- DD Stettler
- DE Meyer
- Diego Fernández Slezak
- DJ Amit
- DJ Felleman
- DJ Freedman
- DJ Freedman
- DS Touretzky
- E Formisano
- E Koechlin
- E Koechlin
- E Salinas
- E Salinas
- EK Miller
- EK Vogel
- EK Vogel
- EL Post
- ET Rolls
- EW Large
- F Kouneiher
- F Vinckier
- G Deco
- G Deco
- G Deco
- G Deco
- G Rainer
- G Wylie
- GE Alexander
- H Bowman
- H Bowman
- H Pashler
- H Pashler
- H Pashler
- H Pashler
- H Pashler
- H Pashler
- IR Olson
- J Bullier
- J Duncan
- J Kawahara
- J Oristaglio
- J Tanji
- JD Cohen
- JD Roitman
- JD Schall
- JD Wallis
- JE Kamienkowski
- JE Laird
- JE Raymond
- JI Gold
- JM Fuster
- JM Hupe
- JR Anderson
- JR Anderson
- JR Stroop
- K Shapiro
- K Whittingstall
- Karl J. Friston
- KF Wong
- KFE Wong
- KM Arnell
- KM Arnell
- KM Arnell
- KS Rockland
- M Graziano
- M Graziano
- M Riesenhuber
- M Sigman
- M Sigman
- M Sigman
- M Watanabe
- M Watanabe
- Mariano Sigman
- MC Smith
- MD Byrne
- ME Mazurek
- MI Posner
- MI Posner
- MM Chun
- MN Shadlen
- MN Shadlen
- MR Nieuwenstein
- MR Nieuwenstein
- N Brunel
- N Fragopanagos
- N Fujii
- N Sigala
- NA Busch
- NK Logothetis
- NP Rougier
- P Jolicoeur
- P Jolicoeur
- P Lakatos
- P Lakatos
- P Sessa
- PE Dux
- PE Dux
- PE Dux
- Pieter R. Roelfsema
- PL Smith
- PR Roelfsema
- PR Roelfsema
- PR Roelfsema
- PR Roelfsema
- PR Roelfsema
- R Dell'acqua
- R Dell'acqua
- R Marois
- R Marois
- R Ratcliff
- R Romo
- R VanRullen
- RC O'Reilly
- RD Luce
- RS Menon
- S Dehaene
- S Dehaene
- S Dehaene
- S Dehaene
- S Dehaene
- S Funahashi
- S Fusi
- S Nieuwenhuis
- S Ullman
- SA Bunge
- SA Hillyard
- SG Kim
- SI Shih
- SJ Luck
- SJ Luck
- SP Wise
- Stanislas Dehaene
- T Jubault
- T Shallice
- T Shallice
- TAW Visser
- V Di Lollo
- VA Lamme
- W Li
- WF Asaad
- XJ Wang
- Y Jiang
- Y Jiang
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2010
- Field of study
The human brain efficiently solves certain operations such as object recognition and categorization through a massively parallel network of dedicated processors. However, human cognition also relies on the ability to perform an arbitrarily large set of tasks by flexibly recombining different processors into a novel chain. This flexibility comes at the cost of a severe slowing down and a seriality of operations (100–500 ms per step). A limit on parallel processing is demonstrated in experimental setups such as the psychological refractory period (PRP) and the attentional blink (AB) in which the processing of an element either significantly delays (PRP) or impedes conscious access (AB) of a second, rapidly presented element. Here we present a spiking-neuron implementation of a cognitive architecture where a large number of local parallel processors assemble together to produce goal-driven behavior. The precise mapping of incoming sensory stimuli onto motor representations relies on a “router” network capable of flexibly interconnecting processors and rapidly changing its configuration from one task to another. Simulations show that, when presented with dual-task stimuli, the network exhibits parallel processing at peripheral sensory levels, a memory buffer capable of keeping the result of sensory processing on hold, and a slow serial performance at the router stage, resulting in a performance bottleneck. The network captures the detailed dynamics of human behavior during dual-task-performance, including both mean RTs and RT distributions, and establishes concrete predictions on neuronal dynamics during dual-task experiments in humans and non-human primates
From fuzzy to annotated semantic web languages
- Author
- A Chortaras
- A Ragone
- A Ragone
- A Ragone
- A Ragone
- A Zimmermann
- B Cuenca-Grau
- C Damásio
- C Hinde
- C Liu
- C Martínez-Cruz
- C Mateis
- C Mateis
- C Meghini
- C-S Lee
- C-S Lee
- CV Damasio
- CV Damásio
- CV Damásio
- CV Damásio
- CV Damásio
- D Dubois
- D Dubois
- D Dubois
- D Guller
- D Guller
- D Sanchez
- D Sánchez
- DJ Abadi
- EP Klement
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Bobillo
- F Klawonn
- FA Lisi
- FA Lisi
- FA Lisi
- G Stoilos
- G Stoilos
- GJ Klir
- H Wang
- H Yasui
- IA Letia
- IJ Pérez
- J Calmet
- J Medina
- J Medina
- J Medina
- JD Ullman
- JF Baldwin
- JF Baldwin
- JJ Lu
- JJ Lu
- JW Lloyd
- K Todorov
- L Paulik
- LA Zadeh
- LV Lakshmanan
- LV Lakshmanan
- LVS Lakshmanan
- M Denecker
- M Denecker
- M Denecker
- M Denecker
- M Eich
- M Emden van
- M Fitting
- M Fitting
- M Fitting
- M Khamsi
- M Khamsi
- M Kifer
- M Kifer
- M Wallace
- MC Fitting
- MC Fitting
- MI Sessa
- N Díaz-Rodríguez
- N Díaz-Rodríguez
- N Lopes
- N Madrid
- P Hájek
- P Hájek
- P Hájek
- P Kulmann
- P Magrez
- P Vojtás
- P Vojtás
- PA Bonatti
- PC Rhodes
- PS Mostert
- R Ebrahim
- R Hähnle
- R Schweimeier
- R-C Chen
- S Borgwardt
- S Borgwardt
- S Colucci
- S Dasiopoulou
- S Dasiopoulou
- S Hölldobler
- S Konstantopoulos
- S Muñoz
- T Lukasiewicz
- T Lukasiewicz
- T Lukasiewicz
- T Lukasiewicz
- T Lukasiewicz
- T Lukasiewicz
- T Lukasiewicz
- T Mailis
- TH Cao
- TT Quan
- TT Quan
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- U Straccia
- V Slavíček
- WC Rounds
- Y Jiang
- Y Jiang
- Y Jiang
- Y Jiang
- Y Jiang
- Y Jiang
- Y Jiang
- Y Loyer
- Y Loyer
- Y Loyer
- Y Loyer
- Y Loyer
- Y Loyer
- Y Loyer
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
The aim of this chapter is to present a detailed, selfcontained and comprehensive account of the state of the art in representing and reasoning with fuzzy knowledge in Semantic Web Languages such as triple languages RDF/RDFS, conceptual languages of the OWL 2 family and rule languages. We further show how one may generalise them to so-called annotation domains, that cover also e.g. temporal and provenance extensions
Lateral cortical Cdca7 expression levels are regulated by Pax6 and influence the production of intermediate progenitors
- Author
- A Louvi
- A Pontious
- A Sessa
- A Stoykova
- AS Fernandez
- BC Lewis
- C Englund
- CU Hellen
- D Mi
- David J. Price
- H Toresson
- J Guiu
- JC Quinn
- JE Prescott
- JH Lui
- John O. Mason
- M Borkowska
- M Manuel
- M Nonaka-Kinoshita
- M Persson
- N Gaiano
- NA Vasistha
- NL Hayes
- P Malatesta
- PE Thijssen
- RC Osthus
- RE Hill
- RF Hevner
- SC Noctor
- SC Noctor
- T Kowalczyk
- T Miyata
- T Takahashi
- TJ Haggerty
- VS Caviness
- W Haubensak
- Y Arai
- Y Goto
- Yu-Ting Huang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/06/2017
- Field of study
Abstract Background We studied whether regulation of Cdca7 (Cell division cycle associated 7) expression by transcription factor Pax6 contributes to Pax6’s cellular actions during corticogenesis. The function of Cdca7 in mediating Pax6’s effects during corticogenesis has not been explored. Pax6 is expressed by radial glial progenitors in the ventricular zone of the embryonic cortical neuroepithelium, where it is required for the development of a normal complement of Tbr2-expressing intermediate progenitor cells in the subventricular zone. Pax6’s expression levels are graded across the ventricular zone, with highest levels laterally where Tbr2-expressing progenitors are generated in greatest numbers at early stages of corticogenesis. Methods We used in situ hybridization and immunohistochemistry to analyse patterns of Cdca7 and Pax6 expression in cortical tissue from wild-type and Pax6 −/− embryos. In each genotype we compared the graded expression of the two genes quantitatively at several ages. To test whether defects in Cdca7 expression in lateral cortical cells might contribute to the cellular defects in this region caused by Pax6 loss, we electroporated a Cdca7 expression vector into wild-type lateral cortex and examined the effect on the production of Tbr2-expressing cells. Results We found that Cdca7 is co-expressed with Pax6 in cortical progenitors, at levels opposite to those of Pax6. Lowest levels of Cdca7 are found in the radial glial progenitors of lateral cortex, where Pax6 levels are highest. Higher levels of Cdca7 are found in ventral telencephalon, where Pax6 levels are low. Loss of Pax6 causes Cdca7 expression to increase in the lateral cortex. Elevating Cdca7 in normal lateral cortical progenitors to levels close to those normally found in ventral telencephalon reduces their production of Tbr2-expressing cells early in lateral cortical formation. Conclusion Our results suggest that Pax6 normally represses Cdca7 expression in the lateral cortex and that repression of Cdca7 in cells of this region is required for their production of a normal complement of Tbr2-expressing intermediate progenitors
Canagliflozin and Cardiovascular and Renal Outcomes in Type 2 Diabetes Mellitus and Chronic Kidney Disease in Primary and Secondary Cardiovascular Prevention Groups
- Author
- Abraham Oomman
- Accini Mendoza Jose Luis
- Agarwal R
- Aggarwal N
- Aizenberg D
- Akiyama Hitoshi
- Alamartine Eric
- Albisu Jp
- Alvarisqueta A
- Amodeo C
- Andreeva V
- Angelova A
- Aoki Hiromi
- Arcos Edgar
- Aronson R
- Asano Akimichi
- Avendano Jorge
- Bajaj H
- Bakris G
- Bartaskova Dagmar
- Bartolacci I
- Barton Petr
- Belobradkova Jana
- Berli Ma
- Bhushan Raju Sree
- Bompoint S
- Bonadonna Riccardo
- Bordonava A
- Borot Sophie
- Bregman R
- Brenner Bm
- Bull S
- Burst Volker
- Calella P
- Canani Lh
- Cannon Cp
- Cantero Mc
- Capuano G
- Cariou Bertrand
- Cartasegna Lr
- Casabella Ts
- Castellino Pietro
- Cercos E
- Ceriello Antonio
- Chacra Ar
- Charytan Dm
- Chen Nan
- Chen Qinkai
- Cherney D
- Cheung Nw
- Chiovato Luca
- Chouinard G
- Colaso Pdg
- Coloma Gc
- Colombo H
- Commendatore V
- Contieri Fld
- Conway J
- Cournoyer S
- Csecsei Gyongyi
- Csiky Botond
- Cuadrado J
- Cuneo Ca
- Cunha Borges Jl
- Cusumano Am
- D'Avila D
- da Costa Faa
- Danos Peter
- Daroza G
- de Almeida Rs
- de Brito Cm
- De Cosmo Salvatore
- de la Fuente Ral
- De Nicola Luca
- De Serres S
- de Souza P
- de Zeeuw D
- Deak Laszlo
- Deboni Lm
- Deepak Dewan
- Derosa Giuseppe
- Di Carlo Alberto
- Di Cianni Graziano
- Diaz Ruiz Jorge Ernesto Andres
- Diego Higuera Juan
- Dimitrov S
- Dohnalova Lenka
- Douthat Wg
- Dran Rd
- Drasnar Tomas
- Droste C
- Dube F
- Dudas Mihaly
- Dussol Bertrand
- Edwards R
- Edwin Fernando M.
- Faghih Markus
- Farias E
- Faulmann Grit
- Fauvel Jean-Pierre
- Felicio Js
- Ferkl Richard
- Fernandez Montenegro Maria Alicia
- Fernandez Mf
- Finkelstein H
- Fragale G
- Fraige F
- Franco Rjd
- Frasca Giovanni
- Fraser I
- Fretes Jo
- Fuiano Giorgio
- Gambaro Giovanni
- Gan Shenglian
- Garcia Ortiz Luis Hernando
- Garcia Nh
- Garibotto Giacomo
- Gastaldi A
- Gelersztein E
- Genadieva V
- Genova-Hristova G
- Giorda Carlo
- Glenny Ja
- Godoy Jorquera Juan Gonzalo
- Goldenberg R
- Gomes M
- Gonzalez Orellana Ronaldo Arturo
- Gonzalez Alexander
- Gonzalez Jp
- Gopalakrishnan Natarajan
- Gourdy Pierre
- Goycoa C
- Gracious Noble
- Greene T
- Greloni Gc
- Guerrero Raa
- Guinsburg A
- Gupta A
- Gupta M
- Hachmann Jct
- Halciakova Katarina
- Haller Hermann
- Hansraj Alva
- Harcsa Eleonora
- Hayashida Cy
- Heerspink Hjl
- Henein S
- Hermida S
- Hernandez Triana Eric
- Hissa Mn
- Hristozov K
- Iitsuka Tadashi
- Ines Molina de Salazar Dora
- Jain Dinesh
- Jardine Mj
- Jerwan-Keim Reinhold
- Johnson D
- Juncos Li
- Kajiyama Shizuo
- Kamenov Z
- Kashine Susumu
- Kawada Toshio
- Keitel E
- Keltai Katalin
- Keresztesi Sandor
- Keshavamurthy C. B.
- Khandwala H
- Khullar Dinesh
- Kiss
- Klein Alexandre
- Klokocnikova Vera
- Klyver Mi
- Kodera Takamoto
- Kohara Sk
- Kong Yaozhong
- Kono Hiroshi
- Konyves Laszlo
- Koundurdjiev A
- Kovar Richard
- Koyama Kazunori
- Kraft F
- Krynski F
- Kumeda Yasuro
- Lanchiotti Pv
- Lastuvka Jiri
- Le Meur Yannick
- Leiter L
- Levin A
- Levin A
- Li Detian
- Li Wenge
- Li Xuemei
- Lin Hongli
- Lisboa Hrk
- Liu Jian
- Lozanov L
- Lu Weiping
- Lukac Martin
- Madore F
- Mah Pm
- Mahaffey Kw
- Major Lajos
- Malaver Natalia
- Malberti Fabio
- Mandreoli Marcora
- Manisha Sahay
- Mannucci Edoardo
- Mao Hong
- Marchetta N
- Margaritov V
- Maxeiner Stephan
- Mcmahon A
- Medina Farina Marcelo
- Mele P
- Milagres R
- Mileder Margit
- Miyauchi Shozo
- Mizuyama Kazuyuki
- Molnar Marta
- Montenegro R
- Moreira Diaz Juan Pablo
- Mucsi Janos
- Muirhead N
- Neal B
- Neto Bg
- Nicholls K
- Nicolai S.
- Niiya Tetsuji
- Nonchev B
- Noronha I
- Novoa Pa
- Oishi Hiroko
- Orio Si
- Oroszlan Tamas
- Orsi Emanuela
- Ory Ivan
- Ota Satoshi
- Otreras F
- Oviedo A
- Packham D
- Panarotto D
- Papini Nr
- Paragh Gyorgy
- Paschen Bjoern
- Paul Haase Franklin
- Pecoits R
- Penfornis Alfred
- Pereira Ma
- Peringat Jayameena
- Perkovic V
- Pesickova Satu
- Peterfai Eva
- Peterka Karel
- Petro Gizella
- Petry Tbz
- Piatti Piermarco
- Pichette V
- Plassmann Georg
- Pollock C
- Portes Ed
- Prasad Narayan
- Proietto J
- Pumprla Jiri
- Rabasa-Lhoret R
- Raduan Ra
- Raffaele P
- Ramirez Roca Luis Alberto
- Ramos Cs
- Rangelov R
- Ren Yan
- Resk Jh
- Revesz Katalin
- Rista L
- Roberts A
- Roger S
- Rose Ludger
- Rosenthal N
- Rosero Ricardo
- Roussel Ronan
- Rychlik Ivan
- Saavedra Gajardo Victor
- Sabbag Arn
- Sakakibara Terue
- Sala J
- Sanches Fcc
- Sanchez Arenales Jose Antonio
- Sanchez Polo Jose Vicente
- Santoro Domenico
- Santos Jc
- Saporito W
- Saraiva Jfk
- Sasso Ferdinando Carlo
- Satyanarayana Rao K.
- Saudek Frantisek
- Saulnier Pierre-Jean
- Schiavi Lb
- Schuch T
- Scotton As
- Serviddio Gaetano
- Sessa H
- Shinkov A.
- Song Weihong
- Sreedhar Reddy
- Sreelatha Melemadathil
- Steele A
- Stella Andrea
- Sudhakar Bhimavarapu
- Sun Jiao
- Sun Lin
- Sun T
- Takacs Robert
- Takai Masahiko
- Tangri N
- Temelkova M
- Terront Lozano Monica Alexandra
- Tesar Vladimir
- Thervet Eric
- Thome F
- Tomonaga Osamu
- Torshizi A
- Trevisan Roberto
- Tsang V
- Tsujimoto Mitsuru
- Tu Ping
- Turatti Laa
- Turcios Juarez Erick
- Ulla Mr
- Valderrama Cometa Luis
- Valdez M
- Valenzuela Alex
- Valis Martin
- Vallejos A
- Vangel Sandor
- Vargas Alonso Ruben Dario
- Vasas Szilard
- Vejar Margarita
- Velichkova E
- Vencio Sac
- Veronelli Anna Maria
- Villarino A
- Villegas Ivan
- Visco Ve
- Vyasam Ramesh Chandra
- Wada Takashi
- Wakasugi Masakiyo
- Wakida Yasushi
- Wang Guixia
- Wassermann A
- Watanabe Takayuki
- Weiner Pavel
- Wheeler Dc
- Woo V
- Yakov A
- Yamada Masayo
- Yamada S
- Yanagida Kazuhiro
- Yanase Toshihiko
- Yang Jinkui
- Yavin Y
- Yin Aiping
- Yu Xueqing
- Yumita Wataru
- Yupanqui Hernan
- Zaidman Cj
- Zalunardo Nadia
- Zanella Mt
- Zanoli Luca
- Zaoui Philippe
- Zemek Stanislav
- Zhang H
- Zhao Minghui
- Zheng Hongguang
- Zinman B
- Zsom Marianna
- Publication venue
- 'Ovid Technologies (Wolters Kluwer Health)'
- Publication date
- 01/01/2019
- Field of study
Background: Canagliflozin reduces the risk of kidney failure in patients with type 2 diabetes mellitus and chronic kidney disease, but effects on specific cardiovascular outcomes are uncertain, as are effects in people without previous cardiovascular disease (primary prevention). Methods: In CREDENCE (Canagliflozin and Renal Events in Diabetes With Established Nephropathy Clinical Evaluation), 4401 participants with type 2 diabetes mellitus and chronic kidney disease were randomly assigned to canagliflozin or placebo on a background of optimized standard of care. Results: Primary prevention participants (n=2181, 49.6%) were younger (61 versus 65 years), were more often female (37% versus 31%), and had shorter duration of diabetes mellitus (15 years versus 16 years) compared with secondary prevention participants (n=2220, 50.4%). Canagliflozin reduced the risk of major cardiovascular events overall (hazard ratio [HR], 0.80 [95% CI, 0.67-0.95]; P=0.01), with consistent reductions in both the primary (HR, 0.68 [95% CI, 0.49-0.94]) and secondary (HR, 0.85 [95% CI, 0.69-1.06]) prevention groups (P for interaction=0.25). Effects were also similar for the components of the composite including cardiovascular death (HR, 0.78 [95% CI, 0.61-1.00]), nonfatal myocardial infarction (HR, 0.81 [95% CI, 0.59-1.10]), and nonfatal stroke (HR, 0.80 [95% CI, 0.56-1.15]). The risk of the primary composite renal outcome and the composite of cardiovascular death or hospitalization for heart failure were also consistently reduced in both the primary and secondary prevention groups (P for interaction >0.5 for each outcome). Conclusions: Canagliflozin significantly reduced major cardiovascular events and kidney failure in patients with type 2 diabetes mellitus and chronic kidney disease, including in participants who did not have previous cardiovascular disease
Search for High-Mass Resonances Decaying to τν in pp Collisions at √s=13 TeV with the ATLAS Detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adye T
- Affolder AA
- Afik Y
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahlen SP
- Ahmadov F
- Aielli G
- Akatsuka S
- Akesson TPA
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexander G
- Alexopoulos T
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alvarez Gonzalez B
- Alvarez Piqueras D
- Alviggi MG
- Amadio BT
- Amaral Coutinho Y
- Ambroz L
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amrouche CS
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Anderson KJ
- Andreazza A
- Andrei V
- Angelidakis S
- Angelozzi I
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antonelli M
- Antrim DJ
- Anulli F
- Aoki M
- Arabidze G
- Arai Y
- Araque JP
- Araujo Ferraz V
- Araujo Pereira R
- Araya S Tapia
- Arce ATH
- Ardell RE
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Armadans R Caminal
- Armbruster AJ
- Armitage LJ
- Arnaez O
- Arnold H
- Arratia M
- Arslan O
- Artamonov A
- Artoni G
- Artz S
- Asai S
- Asbah N
- Ashkenazi A
- Asimakopoulou EM
- Asquith L
- Assamagan K
- Astalos R
- Astigarraga ME Pozo
- Atkin RJ
- Atkinson M
- Atlay NB
- Augsten K
- Avolio G
- Avramidou R
- Axen B
- Ayoub MK
- Azuelos G
- Baas AE
- Baca MJ
- Bachacou H
- Bachas K
- Backes M
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Baines JT
- Bajic M
- Baker OK
- Bakker PJ
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Banas E
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- Bannoura AAE
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- Zwalinski L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2018
- Field of study
A search for high-mass resonances decaying to τν using proton-proton collisions at √s=13 TeV produced by the Large Hadron Collider is presented. Only τ-lepton decays with hadrons in the final state are considered. The data were recorded with the ATLAS detector and correspond to an integrated luminosity of 36.1 fb−1. No statistically significant excess above the standard model expectation is observed; model-independent upper limits are set on the visible τν production cross section. Heavy W′ bosons with masses less than 3.7 TeV in the sequential standard model and masses less than 2.2–3.8 TeV depending on the coupling in the nonuniversal G(221) model are excluded at the 95% credibility level
Search for the direct production of charginos and neutralinos in final states with tau leptons in √s=13 TeV collisions with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abidi SH
- AbouZeid OS
- Abraham NL
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
A search for the direct production of charginos and neutralinos in final states with at least two hadronically decaying tau leptons is presented. The analysis uses a dataset of pp collisions corresponding to an integrated luminosity of 36.1 fb−1, recorded with the ATLAS detector at the Large Hadron Collider at a centre-of-mass energy of 13TeV.Nosignificant deviation from the expected Standard Model background is observed. Limits are derived in scenarios of ˜χ+1 ˜χ−1 pair production and of ˜χ±1 ˜χ02 and ˜χ+1 ˜χ−1 production in simplified models where the neutralinos and charginos decay solely via intermediate left-handed staus and tau sneutrinos, and the mass of the ˜ τL state is set to be halfway between the masses of the ˜χ±1 and the ˜χ01. Chargino masses up to 630 GeV are excluded at 95% confidence level in the scenario of direct production of ˜χ+1 ˜χ−1 for a massless ˜χ01. Common ˜χ±1 and ˜χ02 masses up to 760 GeV are excluded in the case of production of ˜χ±1 ˜χ02 and ˜χ+1 ˜χ−1 assuming a massless ˜χ01. Exclusion limits for additional benchmark scenarios with large and small mass-splitting between the ˜χ±1 and the ˜χ01 are also studied by varying the ˜ τL mass between the masses of the ˜χ±1 and the ˜χ01
Search for anomalous couplings in the W tb vertex from the measurement of double differential angular decay rates of single top quarks produced in the t-channel with the ATLAS detector
- Author
- Aad G
- Abbott B
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 13/10/2015
- Field of study
The electroweak production and subsequent decay of single top quarks is determined by the properties of the Wtb vertex. This vertex can be described by the complex parameters of an effective Lagrangian. An analysis of angular distributions of the decay products of single top quarks produced in the t -channel constrains these parameters simultaneously. The analysis described in this paper uses 4.6 fb−1 of proton-proton collision data at √s =7 TeV collected with the ATLAS detector at the LHC. Two parameters are measured simultaneously in this analysis. The fraction f 1 of decays containing transversely polarised W bosons is measured to be 0.37 ± 0.07 (stat.⊕syst.). The phase δ − between amplitudes for transversely and longitudinally polarised W bosons recoiling against left-handed b-quarks is measured to be −0.014π ± 0.036π (stat.⊕syst.). The correlation in the measurement of these parameters is 0.15. These values result in two-dimensional limits at the 95% confidence level on the ratio of the complex coupling parameters g R and V L, yielding Re[g R /V L] ∈ [−0.36, 0.10] and Im[g R /V L] ∈ [−0.17, 0.23] with a correlation of 0.11. The results are in good agreement with the predictions of the Standard Model
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